ANALYSIS AND FORMATION OF THE ETIOPATHOGENESIS OF DYSBIOSIS IN DENTISTRY (literature review)
Keywords:
dentistry, dental construction materials, diseases of the mucous membrane of the cavities and mouth, dysbiosis, candidiasis, risk factors, oral microfloraAbstract
The formation of normal microbiomaterial of the human oral cavity occurs
on the basis of adhesion and the formation of primary colonies of microorganisms to the
multilayer flat epithelium of the oral cavity. Scientists identify resistant additional and
accidental microflora. A close relationship has been revealed between the formation of
resident microflora of the oral cavity during life and such factors as individual features of
the structure of the mucous membrane, pathological processes on the oral mucosa, nutrition
culture, hygienic aspects, personalized biomechanics of the chewing apparatus, quantitative
and qualitative indicators of saliva, saliva secretion, especially in the presence of orthopedic
structures. Thus, it was found that even with a violation of the immunophysiological
background, the ecosystem of constant normoflora does not participate in the development
of dysbiocinosis and has a low pathogenic potential. The causes of changes in the
micropease and the occurrence of complications are exogenous factors that disrupt
activation by transient and accidental microflora, the ongoing decrease in colonization
protection sharply reduces the resistance of the formation of secondary colonization
(coadgesis) of opportunistic and pathogenic microorganisms. The additional flora is
represented by transient microorganisms coming from the environment, it can be food
products, household contact. In a healthy person, representatives of this group are few (up
to 105 CFU / 1 ml). With a change in the quantity and quality of the composition of the resident bacterial flora, violation of the functional usefulness of the oral cavity, anatomical
integrity, residents of the transient microflora actively form etiopathogenetic links in the
occurrence of pathological processes leading to dysbiosis (including candi dose) of the
mucosa and allows us to consider these conditions generalized rather than local.
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